Dating obese old
People with class I obesity and heart disease do not have greater rates of further heart problems than people of normal weight who also have heart disease.
Complications are either directly caused by obesity or indirectly related through mechanisms sharing a common cause such as a poor diet or a sedentary lifestyle.
As of 2006, more than 41 of these sites on the human genome have been linked to the development of obesity when a favorable environment is present.
People with two copies of the FTO gene (fat mass and obesity associated gene) have been found on average to weigh 3–4 kg more and have a 1.67-fold greater risk of obesity compared with those without the risk allele.
Agricultural policy and techniques in the United States and Europe have led to lower food prices.
In the United States, subsidization of corn, soy, wheat, and rice through the U. farm bill has made the main sources of processed food cheap compared to fruits and vegetables.
A 2006 review identified ten other possible contributors to the recent increase of obesity: (1) insufficient sleep, (2) endocrine disruptors (environmental pollutants that interfere with lipid metabolism), (3) decreased variability in ambient temperature, (4) decreased rates of smoking, because smoking suppresses appetite, (5) increased use of medications that can cause weight gain (e.g., atypical antipsychotics), (6) proportional increases in ethnic and age groups that tend to be heavier, (7) pregnancy at a later age (which may cause susceptibility to obesity in children), (8) epigenetic risk factors passed on generationally, (9) natural selection for higher BMI, and (10) assortative mating leading to increased concentration of obesity risk factors (this would increase the number of obese people by increasing population variance in weight).